A double -covered sword ... "microscopes" protect cells and can cause alzheimers

Researchers at the Advanced Scientific Research Center in the Center for Graduate Studies at New York City University have revealed a strong correlation between cellular pressure in the brain and the development of Alzheimer’s disease. The study published in the journal “Neuron” showed the role of “Microglia”, which is the basic immune cells in the brain, identified as an important player in the preventative and harmful reactions associated with Alzheimer’s disease. The microscope in the central nervous system is the first line of defense against damage and inflammation in the brain and spinal cord, and is similar to the function of other immune cells in the body, such as the Balanti cells in the blood, where it monitors the surrounding environment and searches for any abnormal indicators, such as infection or cellular damage. Microscopic and brain protection when microscopes discover any damage or threat, activate their defense mechanisms, such as swallowing exotic particles such as bacteria or dead cells, or releasing chemicals that help with the body’s reaction to the threat, but sometimes it can cause unexpected damage when it responds to environmental changes. It also plays an important role in the protection of the brain against injuries and infections, as it is involved in disposing of cellular waste, such as damaged proteins and dead cells, which help maintain a healthy nerve environment. It also contributes to the immune response by secreting a group of chemicals that stimulate the immune response and get rid of threats. However, in some cases, microscopes can turn a double sword into a ‘double -weapon’ by contributing to the exacerbation of neurological diseases rather than recovery. And when the brain is exposed to chronic cellular pressure or nerve tension, such as with Alzheimer’s or Parkinson’s patients, the microscopic response and harm, according to the new study, which showed that these cells can also contribute to the aggravation of ‘nervous degeneration’ in Alzheimer’s. In these cases, the microscope becomes excessive and begins to release toxic substances such as toxic phosphorus fats, which damage nerve cells, and it can turn into a type of ‘dark microscope’, causing increased nervous degeneration, contributing to memory loss and poor mental functions. The microscope, which was previously protected the brain, becomes a harmful factor if it cannot distinguish between healthy and damaged cells. In Alzheimer’s disease, harmful microscopic cells are characterized by activating the path of signals linked to cellular pressure, leading to abnormal interaction and contributing to the aggravation of nerve degeneration. The study showed that the harmful cells in Alzheimer’s disease are microscopic cells that occur under the influence of cellular pressure, causing an increase in the secretion of toxic substances, such as toxic fats that harm neurons and other cells in the brain resulting in damage to neurological tangles, which is one of the prominent symptoms in Alzheimer’s. Cellular pressure and nervous degeneration The researchers who use the electronic microscope discovered the accumulation of “dark microscopes” cells in the brain tissue of Alzheimer’s patients. It appears that these cells, associated with cellular pressure and nervous degeneration, were present at the levels of weakness in individuals who were healthy in the same age group. Cellular pressure occurs when cells are exposed to a set of factors that exceed their ability to deal with, leading to changes in cellular functions. In the brain, when the organism is exposed to chronic cellular pressure, neurons and microscopic cells begin to work abnormally, and this condition is directly related to nerve degeneration, which is the gradual decline of neurons found in many neurological diseases such as Alzheimer’s. In Alzheimer’s disease, cellular pressure in neurons and microscopic cells stimulates different cellular signals associated with stress, such as the path of response signals to integrated pressure. This road normally works in cells to protect it from harmful changes, but it can become harmful if it stays for a long time, as happens in Alzheimer’s disease. And when the brain is exposed to cellular pressure, neurons and microscopic cells work through an ISR road, a reaction that includes the production of stress -resistant protein to protect cells from damage. Toxic phosphorus fats said continuous stimulation causes the decline of the function of neurons and microscopes, leading to the production of more toxic substances. Under the ongoing cellular pressure, the microscopic cells that are damaged due to nerve stress, toxic fats, are excreted, such as toxic phosphorus fats that cause damage to the brain cells that play an important role in protecting nerve fibers. Toxic phosphorus fats are fatty compounds that contain a group of phosphate associated with fatty acid and are used in living organisms as part of cellular membranes, but if it is toxic or in a case of self -function, it can become harmful. In most cases, there are toxic phosphorus fats in the cells or in the cell membranes and form an essential part of the formation of the cell membrane that surrounds the cell and separates the internal environment of the cell of the external environment. And when toxic phosphorus fats or the poison accumulates in the cell membrane, the function of the membrane can weaken; This can lead to the leakage of toxic substances in the cells, leading to their decomposition and the toxic phosphorus fats, and the important biochemical reactions found in the cells are hampered. Toxic phosphorus fat can also lead to inflammatory reactions or damage to tissues, and in some cases it leads to damage to biological organs such as the liver, heart and brain. The damage caused by these toxic fats leads to the loss of neurological entanglement, which is a decisive process in memory loss and thinking in Alzheimer’s disease. Toxic phosphorus fats can also be harmful to nerves, as they affect neurological tissues by affecting nervous signal or nerve cell deaths, leading to neurological diseases. In mouse models, the study showed that inhibiting the activation of the ISR road, or the course of fat production, the loss of neuro -transfer substances and the accumulation of toxic protein such as protein “tau”, which provides a promising way to therapeutic intervention. The author, Anna Flori, said the results reveal the decisive relationship between cellular pressure and the toxic effects of the microscope in Alzheimer’s disease. She explained that “the focus of this road can open new horizons for treatment, either by stopping the production of toxic fats or preventing the activation of the harmful microscopes.” This study emphasized the possibility of developing medicines aimed at certain groups of microscopic cells, or their mechanisms associated with cellular pressure. The researchers said that these treatments could help to delay or even reflect the development of Alzheimer’s disease, giving millions of patients and their families hope.