A study that reveals the role of sleep in recovery after heart attacks

A new study from Mount Sinai Hospital in New York showed that the heart communicates with the brain after a heart attack to the desire to sleep, which helps reduce the recovery process and infections. The study published in the “Nature” journal, which is the first of its kind, proves that the heart and brain through the immune system interact to promote sleep as a way of recovery, which supports the idea that a good sleep should be a significant part of the post -heart attack treatment plan, especially in intensive care units. The study shows that the affected heart after a heart attack signals through the immune system sends to the brain to stimulate sleep. Experiments with the mice revealed that deep sleep, or slowly -wave sleeping, increased three times after a heart attack, which helped reduce and recover the heart stress. The microscopic tests of the brain showed that immune cells called ‘mono cells’ moved from blood to the brain, and launched a protein called tumor necrosis factor (TNF), stimulating neurons in the accent area in the brain and stimulating increased sleep. The study also showed that the increase in sleep is an adaptable response to improve the health of the heart to the seizure. Reducing inflammation and good sleep plays a decisive role in reducing inflammation in the body. After the heart attack, the levels of inflammation increase, which is why it can negatively affect the recovery process. Sleep also promotes hormonal balance in the body, including stress hormones such as cortisol, which can lead to extra pressure on the heart and an increase in the risk of heart problems. During sleep, important surgeries occur in the blood vessels; Sleep helps improve vascular elasticity, contributing to the regulation of blood pressure. The researchers notice that drag Alert improves the brain’s communication with the heart to reduce inflammation and increase the recovery faster. The researchers found an increase in the unsaturated cells in the brain after a heart attack, and when the sleeping condition of 80 patients followed after four weeks of Nubia, it was found that those who had a good sleep improved the functions of the heart, while the risks were higher in those who were poor quality of sleep. The study showed that patients who got insufficient sleep were more exposed to relapse, while patients who had sufficient sleep show an improvement in heart function and a haven in inflammation. In another human study, the researchers analyzed the effect of five weeks of sleep on twenty healthy adults, and sleep was monitored using electronic devices, and participants retained notes. During the five -week study period, half of the participants slept seven to eight continuous hours per night, while the other half limited their sleep every night by an hour and a half, or delayed bedtime or woke up early. After the study period, the researchers analyzed the only cells in the blood and found signs of stress and similar inflammatory reactions in the group deprived of sleep, such as those identified in mice. The researchers say that the study reveals new methods that communicate the heart and brain to regulate sleep, which must be absorbed as part of the clinical care of patients after a heart attack.