Myasthenia, under the protection of muscle weakness, is a common name for a number of diseases that harm the area between the neuruscular synapses, when the electrical catalyst is secreted by the nerve, acetylcholine is associated with muscle receptors. This leads to its motivation and the nerve fibers contract, and if the disease is disturbed by this process. The action of muscle delusion is good, and the majority of patients do daily activities well using medication, and the course of the disease is not fixed, and there are periods in which the patient’s condition is worse without any clear explanation. Types of muscle illustration There are different syndromes of the disease and are the following: It is a myasthenia gravis. It is a genetic muscle that occurs as a result of a genetic imbalance in one of the proteins that rides on the muscle nerve, as its work is disturbed. They are a muscle from birth is a temporary disease in infants for mothers who are patient with a domestic muscle, where antibodies reach them through the placenta. It is a muscle in the name of Etone and Lambur is a disease in which a cavity -Cylinder release is caused by the kinetic nerve. This disease can occur in patients with a special type of lung cancer, and can also appear without malignant diseases. It is a muscle due to medication, the disease occurs through medication that can hinder the passage between the nerve and the muscle. There are many medicines like this, the various antibiotics of which are. Symptoms of muscle strength are one of the most important symptoms of muscle weakness: double vision. Poor sound. Problems with swallowing. Pure muscle weakness. The symptoms can be concentrated in one area or in different regions. These symptoms are worse at the time of physical activities, which is why extreme fatigue is a general crucial in muscle weakness, and these symptoms are clearer at the end of the day. The causes and factors of the risk of muscle strength differ from the disease of the disease, as previously mentioned, often caused by the formation of antibodies due to the presence of a genetic defect. Risk factors can start muscle weakness at every age, but it is more common in women of ages between 20 – 40, and the cause of genetic muscle weakness is often when it occurs in early childhood. The complications of muscle weakness are one of the most important complications of muscle weakness: a muscle weakness in which all ribs participate in all voluntary muscles that include breathing muscles. Choosing infection due to medication that weakens the immune system, as the infection itself exacerbates muscle weakness. Permanent muscle weakness, especially in the eyes. Diagnosis of muscle weakness The muscle weakness is diagnosed with various tests of the following: 1. Tennilon test is a substance that leads to high concentration of acetylchlene by impeding the work of the cholinesterase enzyme for a few minutes. The substance is injected into the vein and if there is an improvement in all symptoms, we expect it to be muscle weakness. 2.. A periodic electrical stimulation test and a single -Muscle -fly -fly -Flyer -test. These tests are documented by fatigue or disability in the environment between the nerve and the muscle by registering the electrical event of the muscle due to electric motivation with medium or high frequencies. 3. Anti -blood analysis finds antibodies against acetylchlin receptors at 80% of patients with muscle weakness, and at 10% of the others we may find antibodies against part of the proteins associated with the future, while these proteins cannot be found at 10% of patients. In Etone and Lambur syndrome, antibodies are against calcium channels at the end of the nerve. 4. Other important investigations into muscle weakness are one of the most prominent investigations conducted: examination of the thyroid gland that is effective in 10% of patients with muscle weakness. Investigating the finding of genetic mutations in genetic situations. Treatment of muscle illustration is one of the most important treatments used in muscle weakness. The following: 1. The improvement of electrical compound in the nerves is done by disrupting the work of the Collen Astraz enzyme for a long period, and the most common medicine is pyridotigmin, the treatment is successful for several hours, but it is not the basic mechanism, and it is not necessary. 2.. Weaken the immune system to prevent the production of antibodies. There are two groups of medicines that can suppress the immunity to stop the production of antibodies, namely: Corticosteroids. Toxic remedies for cells, such as: azathioprine, cyclosporine, cyclophosphamide and others. 3.. Modification of the immune system by removing antibodies. The antibodies are removed from the blood system by filtering, or by impeding its effectiveness by injecting a high concentration of immunoglobulin vehicle. Blood purification is the preferred treatment when an attack and a muscle weakness, and when a severe aggravation of the symptoms occurs. 4. Orthodontic gland removed when the timus gland is enlarged and removed. 5. Give medicines that increase the secretion of acetylchlin with ethone Callon syndrome as it helps to get rid of the symptoms of the disease. Prevention of muscle weakness The methods of prevention differ according to the type of muscle weakness. For example, the prevention of muscle is advised to follow: Avoid dealing with sick people. Make sure personal hygiene. Avoid feeling cold or extreme free. Avoid excessive exercise. Learn how to handle stress.
Muscle -illusion: Causes, Symptoms and Treatment
