Zagi Retinal Inflammation: Causes, Symptoms and Treatment

Retinitis pigmentosa – RP under the protection of the Gigneic retinitis over a hundred years ago on a disease during which the chromosome pumps appeared at the base of the eye. The prevailing belief was that the conversation was about inflammation, and it became clear during the 20th century that the disease was hereditary and caused by a genetic defect as it became clear that the disease developed blindness with more than any other genetic disease. There are a group of genetic mutations that can cause the disease, but only some of them are known to today. Sebgi retinitis is a rare disease and the incidence of the disease in the United States is one in almost every 2700 people. The retina contains two types of cells that combine light, namely: sticks and cones, sticks are located around the outer loop of the retina and are activated in the dull light. Most forms of pigment inflammation first affect the sticks, causing the disappearance of the night vision, and the inability to see the side. Concrete is usually located in the middle of the retina, and it helps to see color and fine details, if the disease affects them, the patient slowly loses the central vision and the ability to see colors. The symptoms of the stabbing rhetinitis begin to appear in early childhood, and the most important symptoms associated with the disease are the following: 1. Loss of night vision. Night blindness occurs when the patient can see nothing in the dark, his vision may be normal during the day, but the patient begins to lose night vision, and it takes longer to adapt to the dark. You can stumble into things or have a problem driving at night, and you may find it difficult to see in theaters or other dark rooms. 2.. Gradual loss of peripheral vision is known as this vision as the vision of the tunnel, you may find that you clash with things as you move, because you cannot see the things that exist at the bottom or around you. 3.. Loss of central vision also suffers from central vision problems, and it can make it difficult to perform detailed tasks, such as: reading or sewing. The shrinkage of the field of vision is common, as 94% of patients complain about this, and on the other hand, the effect on the central vision is very diverse. There are patients with gigane, whose central vision is not affected to an advanced generation. On the other hand, there are those who lose their ability to see the central vision in the third decade of their lives. Badia signs change in the eyes according to the period of continuation of the disease, there are no prominent signs in the first decade of life in general, and in the second decade the points and light spots appear in the depth of the retina, and black chromosome points in the network itself. Later, atrophy of the retina appears and shakes in a distinctive bones of the disease. Causes and factors of the risk of stinging retinitis have many causes and factors that lead to inflammation of the chest, and more than 60 genes are responsible for the formation of more than one type of disease transmitted during childhood in the following methods: 1. The birth of a child inherit both copies of the infected genes of parents as it inherits the appearance and symptoms of the disease. And since each child needs two versions of the infected gene to appear, the incidence of the disease for each child here is 25%. 2. Autosomal dominant RP. In this case, the child needs a single copy of the infected gene for the symptoms of the disease, and the possibility of any child in this case is 50% of the possibilities. 3. x Linked gene (X -linked gene) can have sex if the mother is in full health, but transmits the disease to her male children, and many minor symptoms of mothers can occur by a woman from a woman out of 5 women. Most men who inherit the gene of their mothers suffer from severe symptoms, and in these kinds of genes that cause diseases, parents do not send the disease to their children. The complications of the pigmentitis of the pigmentation are the most important complications of the gravity rhetinal inflammation of the following: the incidence of cataracts at an early age. Dissolve the retina. The doctor’s diagnosis can inform the patient about the presence of a chest inflammation after looking at the eye and ordering the following tests: 1. ophthalmoscope. If the patient has a pigmentary retinitis, there will be specific types of dark spots on the retina. 2. Visual field test in this test. The patient looks through a device that is placed on a table at a point in the vision, and during the stare at that point, things or lights will appear on the side, the patient will press a button if he sees it, and the device will create a map from the side of the side that can be seen. 3.. Electroretinogram. The ophthalmologist places a golden chips, or a special adhesive lens on the patient’s eye, and then measures how the retina responds to light flashes? 4. Genetic test is sent a sample DNA to find out the form of the patient’s chest inflammation in the patient. Treatment of gravity -rhetinal inflammation is not yet treatment for the disease, and as the forefront of the 20th century dozens of failed experiments has been performed to treat the disease at the beginning of the placenta implant in the eye stone, performed by a professor called Philatov in Russia, and ends with a series of treatments. 1. Treatments to delay the appearance of visual loss. In contrast, there are types of treatments with a scientific reference that reduces the risk of visual loss, namely: acetazolamide in the late stages of the disease. The small area in the middle of the retina can swell, and it is called the Baqaqiyah, and it can reduce the vision. Acetazolamide can relieve swelling and improve vision. Treatment with vitamin A high doses of vitamin A can help prevent the exacerbation of the disease, but the large amounts of vitamin should be careful, the toxicity can cause, so the doctor’s instructions should be followed on the amount that should be used exactly. Wear sunglasses to reduce liggery, and protect the eye from ultraviolet sunlight that can cause visual loss. Retinal implant, if the patient is infected with later stages of struggling retinitis, it qualifies to plant the retina that provides a partial vision. 2. Treatments that are studied still have serious research to treat gighane retinitis, or a method for actual improvement in the infection of the ability to see, and research is currently taking three different directions: replacement of the affected gene: The replacement process is done by connecting a substance with a right gene and spraying them to the eye or blood. Embryonic cells within the eye: for the purpose of replacing the affected cells. Electronic chip cultivation before or behind the retina: so that the light is taken up and the stimulation is transferred to the optic nerve that bypasses the sick retina. There are good possibilities for the success of one of these trends in the coming years. Preventing the stitch of retinitis can help to consult and test genetically to determine if the child is at risk of developing this disease.